Liz Bode
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Replying to mike m. 10/12/2022 - 15:19
Hi Mike,
I think, if you can from an owner perspective, that this is always a good thing to do. I talk a bit more about it in the feline week, but if you have a breed that has a high chance of HCM such as a Ragdoll or Sphynx then an echo (or brief LA:Ao and HCM check) or NTproBNP would be a sensible plan. Echo would always be first choice but failing that the BNP should at least pick up more severely affected cases.
Liz
Hi Scott,
Yes, this paper is sometimes referred to for the management of pericardial effusions. I think it was the RVC if I remember correctly. To be honest I have never thought or needed to place a pericardial catheter in for a longer period of time than drainage takes. In my mind, if you drain them then only a handful will refill quickly and these tend to be the neoplasia cases. So given it is the minority of cases that refill quickly I think most PE cases don’t warrant the expense/ expertise/ nursing care that placing an in-dwelling catheter would need. Most of my PEs tend to go home after 24 hours. I guess you could consider an in-dwelling catheter in those cases that refill quickly whilst you are waiting for diagnostics to be returned.
Liz
Replying to Nicholas K. 16/11/2022 - 10:48
Hi Nick,
So sorry that this slipped form my radar too! In answer to your echo related questions:
For fractious cats I would use gabapentin initially – it takes the edge off them and has little effect on echo parameters so is fine to use. It will only allow you to echo those cats that are anxious and not the ones that are very aggressive – in my experience you still need something stronger for this. In cases where gaba is not enough I would use butorphanol and alfaxalone – IV if you can get access with gaba but if not IM – only issue is that IM does seem less reliable but it is only a small number of cats where you would need something other than IM butorphanol and alfaxalone.
Liz
Replying to Thaleia M. 27/11/2022 - 19:55
Hello Thaleia,
This is a good question. Cats are so tricky – we always joke that we should echo EVERY cat (or perform NTproBNP) as you never know which one has heart disease and which doesn’t. However, if you have no abnormalities on auscultation at all, and the cat is otherwise healthy, then I wouldn’t be inclined to do anything further. Then if things change later in the cats life you could always perform NT-proBNP at that point (or echo if you can).
Liz 🙂
Hi both,
Great to see you guys on here too 🙂 I hope you get something from it. Last year we had another ESAVS candidate and she really enjoyed it, so fingers crossed!!
Absolutely, ask about other certificates in the UK. I am module coordinator for the Improve Distance learning Cardio Cert and for the Liverpool CertAVP but I have limited knowledge of any others. Sadly, none combine theory with practical echo as far as I am aware.
Liz
Replying to Laura T. 14/11/2022 - 16:07
Hi Laura
Great to have you joining us. I hope that you find the course useful and I look forward to working with you over the coming weeks.
Liz
Replying to Kathryn B. 01/11/2022 - 11:59
Hi Kathryn (and everyone)
I made this little video running through the x-rays, so here it is. Let me know if you have any questions 🙂
Liz
https://drive.google.com/file/d/18AKFZw4OHuoMYZbtNyW7fUjuPaa4ID6o/view?usp=sharing
Replying to Kathryn B. 26/10/2022 - 08:18
In that case it’s this one! Sorry I couldn’t spot where I’d mentioned it.
https://onlinelibrary.wiley.com/doi/10.1111/jvim.15854
Liz
Replying to Kathryn B. 23/10/2022 - 20:25
Hi Kathryn,
Sorry for the delay and it was nice to ‘meet’ you last night. I’ve looked through my slides and my recording and I cannot see where I mention VHS and a paper, sorry! I do mention the VLAS score in association with a paper. It is:
https://avmajournals.avma.org/view/journals/javma/253/8/javma.253.8.1038.xml
It says you can tell if there is LA enlargement with a VLAS of >2.3 in the abstract but in the paper it is more accurate at >3.
In terms of osteomata/ vessels and neoplasia. This can be tricky. Osteomata tend to have a ventral distribution and are mineral opacity whereas vessels and neoplasia are usually soft tissue opacity. Ostemoata are also very small and there tends to be multiple of them. With vessels you can usually trace back to the origin or they overly a vessel as they turn the corner and come towards you. I guess the real solution is that if you are not sure to repeat the radiographs again in 3-4 weeks to see if there are any changes.
Liz
Replying to Sybil Dryburgh 23/10/2022 - 23:14
Hi both
Yes, this group originally did a retrospective study on this (although I’m not sure how you do a retrospective study about applying pressure and the presence or absence of a murmur!). They then went on to prove the concept with this prospective study.
In some cats, usually older and thinner cats, it is possible to place your stethoscope on the right parasternum and cause a low grade systolic murmur. The same goes with echo in some cats. When looking at the right ventricular outflow tract from the right side you can cause a dynamic obstruction due to the pressure of the probe. Another study (before we knew that some of these could be iatrogenic) back in 2004 showed that cats with dynamic right ventricular outflow tract obstruction didn’t have associated pathology or heart disease that would cause it. So, putting two and two together (and hopefully making 4) I think it would be safe to say that these cats probably don’t have pathology but due to their chest conformation you can push the heart against the opposite wall of the chest and deform the right outflow tract resulting in a murmur. When you release the pressure the murmur should disappear. Obviously, if it doesn’t disappear then another cause of the murmur might be present and if you’re not sure then echo is indicated.
I don’t think we know about dogs but I think the majority of dogs have too big a chest for this to be an issue (think how narrow cat chests are compared to a chihuahua even!)
Hopefully this answers your question but let me know if there are any others.
Liz
Replying to Kathryn B. 17/10/2022 - 18:15
That’s so sad! The last Golden I saw was a 3year old with pericardial lymphoma presenting as pericardial effusion, took me completely by surprise and had a similar outcome as they do very badly 🙁 I hope your run of sad cases changes soon.
Replying to Kathryn B. 17/10/2022 - 16:19
Hi Kathryn,
Ooh drainage of big dogs like this can take AGES!!! I’d use a Mila chest drain in these cases as you’ll find them much faster and should reach further into the chest cavity as it probably was the length of your needle that was the limiting factor. You can suture them in for a few days. However, if you’ve drained the chest and it keeps recurring for longer term management I’d use a pleural port ideally (depending on the source of the effusion). A haemorrhagic effusion in a golden retriever doesn’t sound great 🙁Replying to scott@vtx-cpd.com 17/10/2022 - 13:52
Hi Scott
Thanks for this and providing the hydrocodone dose.
I was at a stenting course recently and the course leader used lofenoxal @ 0.2-0.5mg total dose BID as a cough suppressant. Have you used this before? I believe it’s very effective but now needs an import licence?Liz
Replying to Emma Holt 06/10/2022 - 15:49
Hi Emma,
The cut-off for myocarditis is unclear, but I would be more suspicious if the cTNI was >1. I think most cardiologists go by this too. Generally they are well above this in the acute stages. It is good to be mindful that chronic myocarditis (at least in people) may have normal or only mildly elevated cTnI, but they usually have a DCM phenotype.
Liz
Replying to leonique v. 05/10/2022 - 15:33
Hi Leo,
Sure no problem (probably my accent and slurring the words). What we want in pulmonary hypertension is a PDE5 inhibitor as this increases level of nitric oxide resulting in vasodilation of the arteries. This is what sildenafil does in particular within the pulmonary vasculature. Pimobendan is a PDE3 inhibitor and cause arterial dilation via changes to calcium levels in systemic and probably pulmonary vessels. The recent consensus statement on pulmonary hypertension states that pimobendan should be used in cases of post-capillary pulmonary hypertension, ie that caused by left heart disease, but it’s use in pre-capillary hypertension (pul hypertension due to airway disease) has not been studied so it’s use in this situation is unclear. I was referring to cor pulmonale in the lecture – where pulmonary hypertension causes right sided cardiac remodelling and in this instance pimobendan is likely indicated, especially when systolic dysfunction of the right ventricle is present.
Does that make sense?
Liz 🙂 -
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