Gail Smith
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What abt giving it BID Scott? I have seen some vets only subscribe it SID. Interestingly, I take omeoprazole as I have no gall bladder. I asked my GP abt it and he said in humans it is recommended BID
I have been using Aktivait on cat for 3wks now. i think its making a difference. She is brighter, engaging and has a few active moments which are quite amusing. She had starting this yowling for no reason thing which is quite alarming. She has definately stopped doing that as much wuth the Aktivait
i just started mine on Aktivait
she is CKD, had a bilateral thyroidectomy and very arthritic. 19yrs old She is on Gabapentin and low dose metacam. Doing good but seems prone to infections like cystitis
She is very opinionated and has launched a war on a small ball she found behind the sofa. Evertyime she ventues downstais she bats it accross the floor the shouts like a banshee. i think there may be some CDS. but she seems happy
I had a discussion with my vet and we have decided to also try vivitonin. she does this thing where she sits on the stairs and yowls who head off for apparently no reason. We checked her BP yesterday and its fine. She’s been doing it about a year but it was mostly at night before but now seems to be after meals and any time of the day. She has no BP issues so I think its CDS. Not really any other things but she has definitely become more food aggressive (human food), opinionated and demanding and gets really stroppy if she does not get what she wants – she was never like that before.
This is interesting. Im going to try Aktivait with my 19yr old cat. atm I can akin her to a little old lady running around hitting people with her handbag.
Joking aside though she is definitely acting strangely and I wonder about cognitive dysfunction
Just wondering if you could try nutramega supplements?
A few years ago I had similar problems with my cat’s coat and Royal Canin suggested feeding SKIN & COAT
The other option for an old guy is Royal Canin Senior consult Stage 2 – this is formulated for older cats and has some coat support included.I have seen a cat where the catheter detached from the hub after 24hrs. i had attributed it to interferance which is possible BUT after reading this info abt this particulsr catheter i do wonder
Hi Scott
In particular people were discussing benzodiazepams
Other drugs that come to mind were midazolam, ketamine (often used in triple combos for cat anaesthesia)There was particular focus in pre-med preparations that involve a combination of drugs drawn up in the same same syringe
Thanks Scott
My issue with cats is this. dyspnoeic cats get really stressed and I cannot think of a time when a conscious radiograph I possible with these guys, even if they are well enough. Guidance on the EEC courses is hands-off with these types of cats and as little handling as possible under these circumstances until they are stable, which means we need to use other means of diagnostics to get them to that stage. even a TFAST on these guys can be a challenge. On the other hand sedation or anaesthetic drugs may pose some risks in these types of patients.
Im also wondering if the focus from ECC clinicians would lead to this. During my observations the TFAST is used to determine the presence of free fluid prodominently and many people do not feel confident enough to make detailed diagnosis apart from gross anatomy abnormalities. Would this type of assessment be more appropriate after the stabilisation stage by a specialist who would undertake further detailed diagnosis to screen for underlying nature of the heart disease and severity?
Yes, that is along the lines i was thinking Liz, that radiographs are prob not the first thing u’d do but a POCUS is. I am glad i know how to do it but was just thinking abt those patients where it may not be the first port of call. would love to hear what the ECC guys think though
Im thinking of that angry cat struggling to breathe and in heart failure. Radiographs may not be the first thing one reaches to do for it in an ECC setting. Same goes for a dog thats wigging out
Yes, these were post-op colic cases. Seemed like it was a very serious risk in these cases and I saw many DIC cases in horses
Yes, I also wondered if there is a possibility small animal DIC cases are underdiagnosed as well.
I just wondered about how one establishes ‘healthly’ donors? As a nurse we are exposed to a multitude of “nutrition” sins, particularly in 1st opinion practices where the mainstay of nurse clinics are weight clinics. As a result it is clear that there is little understanding of the importance of appropriate nutrition with owners feeding their animals sub-std ‘off the shelf’ diets packed with e numbers and salt, commercial raw diets which (lets face it) demosntrate little explanation as to what is in them. Not to mention the owners who swear blind that an obese Fluffy gets no human food then after the hour long consult it transpires that sausages and bacon are actually on the menu.
The point iof this post is that if we are not giving our animals healthy nutrition, are our donors not at risk of being ‘faecally unhealthy’ without us knowing or without there being a test for it?
Thank you all so much for your responses. I feel less scared about this question in the exam now.
As an aside: I know horses are not small animals but this is interesting
When I was in the US many years ago I ran blood gas/drug screening on the racetrack in Chicago (1995-ish)
In the States they do something called ‘milkshaking’ Basically they add bicarb to about 1-2kg of sugar, mix it into a paste then stomach tube it into the horses pre-race
The theory was that pacing/trotting horses switch to anaerobic respiration at the quarter of a mile point and this ‘concoction’ delays that switch to anaerobic respiration. BUT vets were noticing major issues with thermoregulation/hemostatis so some states banned ‘milkshaking’ Blood was pulled from all horses 6hrs pre-post time and blood gas run (HCO3, Na and pH) If these values were indicative of the milkshaking thing then the horses was re-tested and banned from racing if not satisfied. Different parameters were given to horses receiving Lasix (frusemide) – yes, they administer frusemide to horses that bleed from their lungs when they run (barbaric is it not!!)At the time I never really understood why it was banned but vets reported things like organs bursting and so on – i think at the time they did not really have any scientific basis for why it was bad…just weird stories about horses overheating and collapsing
Now I have been looking at this revision effectively those trainers were deliberately inducing a severe metabolic alkalosis and actually instead of maximising aerobic respiration they were in fact impairing it further. for the bleeders receiving diuretics pre-post time it was even worse
I just wondered….I assume damage to kidneys, brain and cardiac muscle was occuring here?
Not a blood gas thing but one of my jobs there was to tongue swab horses in the paddock for Cocaine. CRAZY HUH
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