Sara: just a challenge to your post: =)
How would we know that Metabolic acidosis isn’t due to Lactic acidosis with hyperlaceramia or DKA, or toxin: Ethylene Glycol , uremic acidosis vs diarrhoea, renal tubular necrosis if we don’t have access to anion gap?
and how do we know that the high lactate isn’t due to hypovolemia until we try to fluid resuscitate the patient and recheck? eg secondary to complicated DKA. 🙂
